Etiology of Sickle Cell Crisis

The clinical manifestations of sickle cell anemiaduring the neonatal period and early part of
vary markedly in severity and frequency. Theinfancy, it may not be recognized until the toddler
most acute symptoms of the disease occurand preschool period. It is frequently first
during periods of exacerbation called crises. Therediagnosed during a crisis, after an acute upper
are four types of episodic crises vaso-occlusive,respiratory or gastrointestinal infection. Crisis, after
splenic sequestion, aplastic, and hyperhemolytican acute upper respiratory or gastrointestinal
crises.infection.
Vaso-occlussive crises are the most common andSeveral tests are available for detecting sickle cell
the only painful ones. They are the result ofanemia. Although most of the routine
sickled cells obstructing the blood vessels, causinghematological tests described are done primarily
occlusion, ischemia, and potentially necrosis. Theto evaluated the anemia, this discussion focuses
major symptoms of this crisis are fever, acuteon the four tests specifically used to detect the
abdominal pain from visceral hypoxia, hand-foothomozygous or heterozygous for of the disease.
syndrome, and arthralgia, without an exacerbationFor screening purposes either the sickling test of
of anemia.the sickledex is commonly used. If the test is
Splenic sequestration crises are caused by thepositive, hemoglobin electrophoresis is necessary
spleen sequestering (pooling) large quantities ofto distinguish between those children with the trait
blood, causing a precipitous drop in blood volumeand those with the disease. Screening for sickle
and ultimately shock. The crisis may be acute orcell trait has become a controversial subject,
chronic. The chronic manifestation is termedespecially among the black community, since
functional asplenia. The acute form occurs mostthere is no method of preventing the disease
commonly in children between 8 months and 5other than selective birth procedures.
years of age and may result in death fromStained blood smear
profound anemia and cardiovascular collapse.Examination of the stained smear of blood may
Aplastic crisis is diminished red blood cellreveal a few sickled red blood cells. However,
production, usually triggered by a viral or othersince the erythrocyte assumes its normal discoid
infection. When superimposed on the existing rapidshape under adequate oxygenation, non-sickled
destruction of red blood cells, a profound anemiacells may be present even in the homozygous
results.form of the disease. Whenever sickle cells are
Another type of bone marrow crisis isfound, the diagnosis is usually positive for sickle
megaloblastic anemia, which is attributed to ancell anemia, not sickle cell trait.
excess nutritional need for folic acid/ or vitaminSickling test (sickle cell slide preparation)
B12 during periods of pronounced erythropoiesis.The simplest method to detect sickling is to place
Since infection is not always antecedent toa drop of blood on a slide and cover it with a
aplastic or hypoplastic crises, it is possible that folicsealed cover slip to produce de-oxygenation.
acid deficiency is a causative agent.Eventually sickling of the red blood cell occurs.
Hyperhemolytic crisis occurs when there is anUnfortunately this test may take several hours
even greater rate of red blood cell destruction. Itbefore results are obtained, and it is not specific
is characterized by anemia, jaundice, andfor the disease or trait. False negatives for the
reticulocytosis. It is a rare complication andtrait can occur if the blood contains a very small
frequently suggests other coexisting abnormalities,amount of hemoglobin S.
such as glucose-6-phosphate dehydrogenaseHemoglobin electrophoresis ("fingerprinting")
deficiency, which is also common in black persons.In this test, the blood is specially prepared and
Diagnostic evaluationseparated into various hemoglobin by high-voltage
Although sickle cell anemia has been reportedelectrophoresis.